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MS causes the immune system to attack myelin — the insulating sheath around nerve fibers — producing progressive disability. Current treatments slow the immune attack but don't repair existing damage. Distributed simulation models oligodendrocyte precursor cell behavior under different molecular conditions, identifying the signals that trigger natural remyelination. If these signals can be pharmacologically activated, damaged myelin could regenerate in MS patients. Supported by MS Society Research and Cambridge Neuroscience.
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