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Cystic fibrosis is caused by CFTR protein misfolding — producing thick mucus that destroys lungs and digestive organs. Over 2,000 CFTR mutation variants exist; most have no approved therapy because each patient population is too small to fund clinical trials. Distributed simulation models how corrector compounds interact with each rare mutation variant, creating a computational therapy atlas accessible to researchers worldwide. Every corrector-mutation interaction your computer models narrows the path to treatment for patients who currently have none. Supported by the Cystic Fibrosis Foundation and Stanford Biochemistry.
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